We will discuss current knowledge, the underlying pathways, and regulation via complex interplay between internal physiological factors and external therapeutic modulators targeting intestinal cholesterol absorption. LDL-cholesterol at a mean dose of 2. Excreted as cholesterol or bile acid. Cholesterol and other sterols bind directly to several immune receptors, especially in macrophages and T cells, and dynamic changes in cholesterol biosynthesis impact directly upon innate and adaptive immune responses, such that functional coupling between sterol metabolism and immunity has implications for health and disease. An overview of cholesterol absorption is the focus of this chapter, but due to the unquestionable importance of an integrative view, the relationship with cholesterol synthesis and fecal excretion is also reviewed. CETP, a serum protein, facilitates the exchange of triacylglycerol and cholesterol esters between HDL and LDL. The movement of cholesterol can be manifest as exchange, in which case no net movement of cholesterol occurs, or how cholesterol excreted from body a flux of cholesterol from one membrane to another.
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